Part of our aging process is tucked away in the complex system of our chromosomes. Each chromosome is made of protein and a single molecule of ‘Deoxyribonudeic Acid’ or DNA – the treasure-trove of information that makes us related to our parents yet creates our unique features. At each tip of these busy twisted ladders are protective caps known as Telomeres, These unique structures also contain an array of highly repeated DNA sequences and specific binding proteins.
However, cellular activity has a devastating effect on these tiny tips. Every time cells divide, the telomeres shorten and healthy cell renewal becomes less and less.
Short Telomeres – big aging impact
Research has found the shortening of the telomere is the trigger to the aging process. It supposedly causes our cells to get older and slowly shut down their essential activity. This inefficient gene function creates problems with inflammation, oxidative stress and immune cell aging. Essential proteins don’t get produced and, eventually the aging process starts its downhill roller coaster journey through your body.
Skin cells ceases to refresh and the tight surface skin we have when young starts to become loose. Those bright eyes dim as the telomeres cannot keep up the cellular dividing. Our immune system finds it too weary to keep on battling and we become more vulnerable to everyday problems such as colds and flu, and have less protection against more serious contagious diseases.
Short telomeres are also thought to cause other diseases that we associate with aging, such as osteoarthritis, diabetes, and cardiovascular disease.
Telomerase to the rescue!
Research found that an enzyme called Telomerase packs a mighty punch when it comes to the Telomere. It is perhaps, part two of the Telomere puzzle.
In 1973, Soviet biologist Alexey Oloynikov suggested there was some kind of the ‘compensatory mechanism’ for the devastating telomere shortening. His theory was found to be sound in 1984 with the discover of Telomerase by Carl W. Greider and Elizabeth Blackburn. Further research by their group proved so outstanding as to be awarded a Noble Prize.
With the presence of Telomerase, each dividing cell can easily replace the lost ‘bit’ of DNA caused by the shortening of the Telomere. Single cells can then divide unbounded and do their job of vital body management.
Telomerase could be called the keeper and repairer of the Telomere!
Fountain of Youth
So when the Telomere is in fine shape again, the aging process may have to take a back seat! When cells continue to keep stay strong, our skin improves its elasticity, libido perks up and our immune system works more efficiently.
Activating this super enzyme Telomerase may just be the tip of an exciting mountain of anti-aging secrets.
CA-98 Advanced
CA-98‘s Cycloastragenol (CA) attacks the issues associated with aging at their root and is believed to produce a great number of beneficial health effects.
CA-98 contains pure Cycloastragenol (CA) formulated via proprietary extract methods containing 98% Cycloastragenol (CA-98). The therapeutic targeting of Cycloastragenol for telomerase activation in relation to ant-aging has been proven by various clinical trials.
With the aid of CA-98’s Cycloastragenol (CA), telomerase activity is turned on, which helps make human cells live longer and reduces the loss of cells.
Keeping the Telomere long and healthy could mean a long and healthy life for all.
Also Read: Stress Recovery and Telomere Length-How Closely are they Related
